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Recent research has shown that an endothelial-specific glycocalyx component, endomucin (EMCN), is highly expressed in the glomerular endothelium and plays a key role supporting the normal structure and function of the glomerular filtration barrier by maintaining the tight junctions, homeostasis of the glomerular endothelium, and function of podocytes (81)

Notably, these pathways demonstrate significant cross-talk, where dysfunction in one system often amplifies perturbations in others

Though the results are inconsistent from population studies, an interconnection between the pathogenesis and catalase may still be possible as scattered demonstrations are reported in the literature