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Considering its safe profile as a bioactive compound [163], AST may become an effective and non-toxic treatment for these GI diseases

SAMe depletion in AD animal models and in vitro has been shown to perturb DNA methylation with resultant increases in the expression of Amyloid Beta Precursor Protein (APP), Presenilin-1 (PS1) and Beta-Secretase 1 (BACE1) genes, stimulating A production and neurodegeneration (13-16)