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Oxidative stress can result either from the excessive production of reactive oxygen species, which might be anticipated at times of glutamatergic over-activity, or from a reduction in oxidative defences, and can damage cell structures

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For further improvement of these in vitro models, implementation of fluid flow and vasculature is crucial, not only to increase maturation of the kidney cells but also to provide an environment that more closely resembles the in vivo situation

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