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2f), demonstrating that AKT is the proximal kinase responsible for the inflammation signal-specific EPRS1 modification

doi: 10.1152/physrev.00012.2004 12 CummingsDEFrayoRSMarmonierCAubertRChapelotD

Relevant to the mechanism of PKC downregulation by antidepressants and valproate (see below), acute lithium use mimics the effect of phorbol esters with resultant activation of PKC (energizing immediate early genes such as c-fos), while chronic lithium use attenuates the phorbol ester action on PKC, down-regulating the enzyme (Manji and Lenox, 1994)

The inflammation can occur suddenly and without warning, and often with no related symptoms

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