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These metabolites also reprogram lipid metabolism by downregulating SREBP-1c and upregulating CPT-1-mediated fatty acid -oxidation, thereby reducing hepatic lipid deposition (Nanji et al., 2003)

Notably, because the cell metabolism also produces H 2 O 2 , the MSCs+H 2 O 2 +Ru-hydroxide can maintain a relatively stable concentration of ~0.38 M H 2 O 2 , which is below the 1 M threshold and comparable to the level in the bare MSCs group without any treatment

The reduced dihydrolipoate residue is then oxidized by FAD and the liberated electrons are passed to NAD yielding NADH

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