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Inhibition of pancreatic EZH2 restores progenitor insulin in T1D donor

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To protect themselves from irreversible damage and prevent the cell from undergoing apoptosis (programmed cell death), the mitochondria initiate a defensive mechanism known as the Cell Danger Response (CDR)

Consequently, activated PTGER2 inhibits TGF- signaling pathway to negatively affect migration and proliferation of myofibroblasts, resulting in a diminution in fibrosis (Zhang et al., 2019b)