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However, no improvement in clinical assessment, tissue pathology, differences in vessel number, or laminin expression in the lesion was observed with chronic drug administration

doi:10.1042/bj20031975 34

Considering that CHAC1 is responsible for glutathione degradation and GLRX is the most well-studied catalytic enzyme for deglutathionylation, we examined the expression levels of CHAC1 and GLRX in these cells