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FIGURE 3 The post-exercise blood lactate levels detected in rat models subjected to exhaustive swimming or treadmill tests showed that acute or short-term peptide supplementation (e.g., at dosages of 200600 mg/kg/day) was able to greatly reduce lactate accumulation when measured immediately post-exercise (61), interfering with reducing the oxidative injury of muscle tissue during maximal exercise

Here we used such iPSC-derived complex liver organoids to assess the specific impact of ALGS-causing mutations on parenchymal and non-parenchymal liver cells, their role on bile duct formation and survival, and the interactions between signaling pathways suspected to play a role in ALGS pathophysiology

On a previous paper using an inflammatory model we used 600 mg/kg and 300 mg/kg and demonstrated that doses of 300 mg/kg were adequate for neuroprotection 17

The autophagic degradation of BMAL1/ARNTL mediated by the cargo receptor SQSTM1/p62 (sequestosome1) is reported to promote lipid peroxidation and ferroptosis [106, 374]