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This first-pass mechanism of periportal toxicity for selected compounds, including hepatotoxicants that primarily affect centrilobular hepatocytes, could result from deficient cytoprotective capacity secondary to depletion of phase II conjugating enzymes in addition to being a consequence of a higher exposure gradient to these upstream hepatocytes

However, our study was conducted in winter time to ensure exposure to high PM2.5 concentration, resulting in relative short-term exposure

Update on bone anabolics in osteoporosis treatment: rationale, current status, and perspectives

Drugs affecting homocysteine metabolism: impact on cardiovascular risk

Bothwell M (1995) Functional interactions of neurotrophins and neurotrophin receptors