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APOL1 protein promotes kidney injury by depolarizing the autophagic membrane to inhibit the fusion of autophagosomes and lysosomes[117]

3e), and increased proliferation and reduced differentiation of P10 OPCs in the IL1-model (Supplementary Fig

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Additionally, GSH levels in tumor tissues decreased in the GGCT knockdown group but were restored by NAC, indicating that NAC could elevate the level of GSH by scavenging ROS (Fig