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In addition, dihydroartemisinin increased the expression of M1 phenotype-related molecules (CD86, iNOS, and Cox-2) and decreased that of M2 phenotype-related molecules (CD206 and Arg-1)

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This result implies that MT interacts with other signal pathways as a signaling molecule, activating the TOR and calmodulin signal pathways, while inhibiting the MAPK signal pathway

Comparative GH Peptide Research: Enables comparisons between single-peptide, dual-peptide, and multi-peptide GH signaling systems

This review systematically describes the metabolic pathways of ferroptosis and summarizes the mechanisms of action of natural and synthetic small-molecule inhibitors of ferroptosis as well as their applications in disease