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A significant elevation of pro-apoptotic Bax protein levels was detected in Zn-supplemented groups, whereas anti-apoptotic Bcl-2 expression was markedly reduced as quantified by immunoblotting data analysis (Fig

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All contribute to hyperammonemia and endotoxemia, and together with the cause of the liver disease itself, promote anabolic resistance, in which the nutrients and physical activity are incapable of increasing protein synthesis and decreasing proteolysis.44 Once the liver is unable to metabolize ammonia, it can only be done by astrocytes and muscle cells, converting ammonia into glutamine through the glutamine synthetase enzyme

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