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By supporting mitochondria, it enhances cellular energy metabolism

BPC-157 is reported to promote angiogenesis, in part through upregulation of vascular endothelial growth factor receptor 2 (VEGFR2) and the nitric-oxide (Akt-eNOS) pathway, and to accelerate the migration and survival of fibroblasts

During the intermediate stage, cumulative oxidative injury amplifies lipid peroxidation through ACSL4/ALOX15-dependent pathways, whereas GSH depletion limits GPX4 activity