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These seemingly counter-intuitive approaches of depleting or overdosing androgens were based on the long-recognized biphasic response of CaP cells to androgens in which low doses stimulate cell proliferation and high doses inhibit growth while inducing differentiation [21,22,23]

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Fatty Acid Transport Protein 2, implicated in arachidonic acid uptake and prostaglandin E2 activation linked to tumor progression, presents a promising avenue for investigating the downregulation of this transporter as a means to potentially decelerate COPD progression (Zhou et al