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Conversely, Nrf2 has been documented to reduce NOX activation and inhibit protein kinase C, consequently leading to a decrease in ROS levels and production in Nrf2-KO mice due to elevated antioxidant GSH levels, highlighting its role in mitigating oxidative stress

This modification improved the hosts tolerance to the immune response against LEH [105, 106], delayed its phagocytic clearance by monocytes, and increased the mean residence time in circulation

106 found that tumor necrosis factor (TNF)- was capable of activating RIPK1, promoting the phosphorylation of RIPK3, and thus forming RIPK1RIPK3 complex, named a necrosome