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First, although SLC30A10 mediates the release of Mn from the brain and SLC39A8 facilitates its uptake 312 , the precise mechanisms that regulate neuronal Mn transport and accumulation remain poorly understood

Recent research suggests that mitochondria contribute significantly to the treatment resistance of PDAC by influencing apoptosis, metabolism, mitochondrial DNA (mtDNA) processes, and mitochondrial dynamics [11, 12]

In glaucoma, the increased levels of A, p-tau, and amyloid precursor protein initiate an inflammatory response, leading to cytokine secretion and iron overload