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Subsequently, we first characterized the direct m 6 A modification target of IGF2BP3 in ovarian cancer as CACNA1A, which was in turn confirmed to promote ovarian cancer proliferation via suppressing Ca 2+ -regulated ferroptosis in our study

Fasting GH-concentrations were reported to be normal or even increased in CKD patients, caused by a prolonged plasma GH half-life, due to reduced GH clearance by the kidneys [42, 155]

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