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Notwithstanding these limitations, findings from our study showing increased lean mass preservation after apitegromab treatment are in alignment with the well-established myostatin biology as well as evidence in preclinical models and consistent clinical findings in SMA studies 9,16

We found that G-6-P administration had no effect on cell death and the levels of p-S166 RIPK1 in HK2-deficient MEFs stimulated with TNF alone or TNF/CHX (Supplementary Fig

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N-terminal hexenoyl group, C-terminal amidation) give it resistance to DPP-IV and peptidase degradation, extending its half-life compared to native GHRH