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Pre-clinical and cosmetic-research data describe increased collagen and elastin synthesis, fibroblast proliferation, improved barrier-protein expression, and reduced markers of oxidative stress following GHK-Cu exposure

This impairs homologous recombination (HR)-mediated DNA repair, thereby contributing to carcinogenesis

Additionally, SB treatment suppressed the production of inflammatory markers IL-1 and chemokine CXCL10, and blocked ischemia-elicited upregulation of COX-2 [17, 18]

however, there are some indications provided by in vitro assays with pharmacological inhibitors and silencing approaches, suggesting that this enzyme can be critical for cancer cell proliferation, bioenergetics, and cell signaling (125)

L., Chen, X