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Protection from tumor necrosis factor-mediated cytolysis by overexpression of plasminogen activator inhibitor type-2

FIGURE 2 Coincided with reduced AF inducibility, LCA supplementation showed significant capacity to shrink LA, attenuate cellular hypertrophy, gap junction disturbance, and fibrosis while challenged by a long-term HFD (Figure 2), indicating FAO promotion via LCA supplementation constrained the AF substrates in obese mice

doi: 10.1038/s41577-021-00639-3 12 KelleyCEBrownAJDiehlAMSetjiTL

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