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10.1074/jbc.m110.119131 145 YuanX.CongY.HaoJ.ShanY.ZhaoZ.WangS.et al (2004)

doi: 10.1038/s41568-018-0007-6 22 ZhaoZChenXDowbajAMSljukicABratlieKLinLet al

In paracetamol (acetaminophen) overdose, toxic metabolite NAPQI depletes GSH

Nrf2 also promotes the production of heme oxygenase-1 (HO-1) to protect hepatocytes from oxidative stress, and HO-1 attenuates alcohol-induced liver damage by suppressing inflammatory responses [11]