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This multi-target mechanism positions it as a promising dietary intervention, in contrast to high-fat diets that exacerbate gut dysfunction (142, 143)

It prevents the generation of free radicals in chemical toxin metabolism in the liver

To induce overexpression of LOXL2 or LOXL2-DM, cells were transduced with adenovirus (100 MOI for both AdLOXL2 and AdLOXL2-DM) for 24 h before initiating the activity assay

the molecular mechanisms of activation of the pro-inflammatory response caused by bacterial invasion through the TLR4-induced TIRAP/MyD88 and TRAM/TRIF signaling cascades

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