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NSAID-associated GI damage is the most common drug-induced GI pathology, making animal models of NSAID gut damage clinically relevant

CFTR gene mutations and polymorphism are associated with non-obstructive azoospermia: from case-control study

p38alpha MAPK-mediated induction and interaction of FOXO3a and p53 contribute to the inhibited-growth and induced-apoptosis of human lung adenocarcinoma cells by berberine