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GluN2B is more abundant in the Bergmann glia cells (Luque and Richards, 1995), whereas the GluN2C is predominantly expressed in the telencephalon glial cells (Alsaad et al., In vivo evaluation of the GluN2C NMDA subunit localization showed that parvalbumin-positive neurons in the globus pallidus, ventral pallidum, and substantia nigra express this subunit, whereas GluN2C in the cortex, striatum, hippocampus, and amygdala colocalizes with astrocytes markers (Ravikrishnan et al., 2018)

Mice treated with GHK showed little learning impairment after sleep deprivation in line with saline-treated mice that were not sleep deprived, and significantly less than sleep-deprived mice treated with saline (Figure 1)

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