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Therefore, sphingolipid-autophagy crosstalk constitutes a pivotal regulatory nexus governing Treg stability, whose diabetic disruption propagates T1DM immunopathology by crippling immunosuppressive circuits, while pharmacologically restoring this axis emerges as a strategic therapeutic paradigm for re-establishing immune tolerance (Figure 5)

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The cells in the hepatocyte zone also exhibited activation of EGFR and phosphorylated-S6, both play a role in pathways relevant for proliferation, differentiation, and survival 18,60 , and liver regeneration 19

A., Ycaza, J