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Description

By enhancing ER protein-folding capacity, reducing the accumulation of misfolded proteins, and activating adaptive signaling programs, the UPR allows cancer cells to survive and proliferate in hostile microenvironments that would otherwise compromise cellular viability

10.1016/s0896-6273(04)00153-9 49 RobertsV

Enhanced expression of egr-1 was combined with a multitude of pathologies (i.e., atherosclerosis [159], stenosed calcific valvular disease [161], cardiac hypertrophy [162,163], cerebral ischemia [164], and tumor progression [158,165,166,167])