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10.1016/j.procbio.2012.05.010 Process Biochem

These results suggested that chronic hypoxia promoted the expression of Acer2 by activating HIF-2

Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stress

Integrating these emerging yet scattered observations, we propose a therapeutic strategy that exploits metabolic vulnerabilities to exacerbate genome instability