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Researchers propose that the peptide's brief presence is sufficient to trigger a cascade of gene expression changesincluding activation of transcription factors c-Fos, c-Jun, and Egr-1that initiate self-sustaining healing processes well beyond the peptide's physical presence

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These results validated that ZZW-115 worked efficiently in blocking the nuclear translocation of NUPR1 in our culture model and that inhibition of NUPR1 nuclear translocation induces cell death in phBECs, indicating an important protective role in bronchial epithelial cells