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469 Meanwhile, the NAMPT is also a target of FoxO transcription factors that control the NAD + signaling in the regulation of hepatic triglyceride homeostasis

The transition from notochordal cells to chondrocyte-like cells is currently debated, with some evidence that notochordal cells are completely replaced with chondrocyte-like cells in certain model systems while other studies show retention of a population of notochordal cells past early developmental stages (Minogue et al., 2010

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Chronic deficiency may elevate the baseline risk of ischemia by impairing vascular elasticity (via compromised LOX function) and promoting platelet aggregation [162]

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