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As crucial regulators of local immune responses and tissue repair, macrophages in diabetic wounds show dysregulated switching from M1 to M2, resulting in a poor regenerative environment, exacerbated tissue damage, and delayed wound healing 63

BPC-157 also enhances growth hormone receptor expression in fibroblasts and promotes FAK-paxillin pathway activation critical for cellular adhesion, migration, proliferation, and survival during tissue repair processes

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doi:10.1038/sj.cdd.4401837