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10.1093/carcin/21.3.361 110 MatschkeJ.RiffkinH.KleinD.HandrickR.LdemannL.MetzenE.et al

However, under the MDD pathological condition, due to the decreased expression of EAATs in astrocytes, excessive glutamate in the synaptic cleft activates synaptic mGluRs, which leads to neuronal excitotoxicity

Cholinergic Signaling Controls Conditioned Fear Behaviors and Enhances Plasticity of Cortical-Amygdala Circuits

Specifically, IL-4 promotes an anti-inflammatory phenotype by upregulating Arg1 [88], whereas IFN- induces a proinflammatory state, leading to the release of TNF- and IL-6 and the upregulation of MHC-II, which contributes to neuroinflammation [89]