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Once activated by oxidative stress, NRF2 translocates to the nucleus where it binds to antioxidant response elements (ARE) in the promoter region of genes involved in redox regulation, xenobiotic metabolism, DNA repair, apoptosis, proteostasis, and regulation of inflammation [9]

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demonstrated that the co-transplantation of human adipose tissue-derived MSCs with MCs extracted from foreskin, or the pre-culturing of MCs alongside adipose tissue-derived MSCs prior to transplantation, resulted in an increased survival rate of MCs in nude mice and Sprague-Dawley rats, without any observed inflammatory infiltration of the skin [155]

However, because of the episomal nature of the adenoviral and AAV vectors after cell entry, these viral vectors are most suited for therapeutic applications requiring only transient expression of the transgene (Russell, 2000, J

Epigenetic dysregulation of epstein-barr virus latency and development of autoimmune disease