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In models, moderate ROS levels in HepG2 and Huh7 cells activate ERK and AKT, leading to the transcriptional induction of mesenchymal markers (Snail, vimentin) and matrix metalloproteinases (MMP-2/9), while concurrently repressing epithelial E-cadherin [91]

Montazeri-Najafabady N, Dabbaghmanesh MH, Namavar Jahromi B, Chatrabnous N, Chatrsimin F

They are linked through overlapping pathways including NF-B activation, elevated TNF-, and IL-6 that drive both gut permeability and systemic insulin resistance.13 Peptide therapies for insulin resistance increasingly recognize this connection

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[12] Gastric acid-suppressing agents (proton-pump inhibitors and H-receptor antagonists) diminish dietary B12 liberation from protein complexes, a pharmacodynamic interaction that can exacerbate underlying deficiency