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doi: 10.2337/db05-1646 56 StovingRKChenJWGlintborgDBrixenKFlyvbjergAHorderKet al

The increased oxidative environment resulting from NOX activity and mtROS production leads to GSH depletion

In vitro experiments demonstrated that agrin treatment significantly downregulated PDGFR and claudin-5 expression in a blood-brain barrier co-culture model comprising human brain microvascular endothelial cells and pericytes, suggesting that agrin may induce pericyte death and impair the integrity of the blood-brain barrier

In contrast, a study identified phosphorylation of BID by JNK at T59 prevents caspase 8 cleavage, but despite the lack of cleavage this drives apoptosis (Prakasam et al., 2014)

MishraAPrakashSSreenivasVDasTKAhujaVGuptaSDet al