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S1 protein recognizes, through a receptor-binding domain (RBD), the human angiotensin-converting enzyme 2 (ACE2) as its main receptor [8]

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Increased efflux of oxidized glutathione (GSSG) causes glutathione depletion and potentially diminishes antioxidant defense in sickle erythrocytes

Key questions including the existence of lactyl-CoA in vivo, the physiological relevance of non-enzymatic lactylation, and the identification of specific reader proteins must be resolved before lactylation can be fully established as a stable and universal regulatory mechanism