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Description

51 CPhar acts as a binding partner of DDX17 which sequesters C/EBP, a key transcription factor involved in exercise-induced physiological cardiac hypertrophy, thus leading to reduced transcription of ATF7

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The molecular mechanism by which CTSB degrades FPN to disrupt macrophage iron homeostasis and promote the progression of atherosclerosis

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Woods, A., Garvican-Lewis, L

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