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While it has not been conclusively established whether this dysfunction is a cause or consequence of the pathological processes, growing evidence suggests that reduced proteasomal activity may contribute to the accumulation of toxic protein aggregates and the progression of neurodegeneration

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In this B12 article, I am focusing on the vitamin itself, because it deserves dedicated treatment given how commonly it is deficient in post-weight-loss patients and how significant the consequences of that deficiency can be

Any observations regarding changes in body weight or metabolic parameters that arise within controlled experimental protocols should not be generalized beyond those research settings

D+Q treatment mitigated pathogenic ventricle enlargement, aberrant cerebral blood flow and white matter hyperintensity pathology (Figure 2ac, adapted from (21))