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[DOI] [PubMed] [Google Scholar] 226.Gado F., Ferrisi R., Polini B., Mohamed K.A., Ricardi C., Lucarini E., Carpi S., Domenichini F., Stevenson L.A., Rapposelli S., et al

Steinherz PG, Redner A, Haimi J, Meyers P, Andreeff M

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In mouse models, mitochondrial dysfunction induced by CDN administration and energy and nutrient deficiencies caused by Cu consumption, elevated oxidative stress, and mitochondrial membrane rupture contribute to the apoptosis of TNBC cells, inhibit tumor growth, and significantly improve the survival rate

Selvin, Elizabeth et al