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A central mechanism driving such pathological scarring is the persistent activation of the transforming growth factor-beta (TGF- )/Smad signaling pathway, which continuously stimulates fibroblasts and myofibroblasts, resulting in excessive collagen deposition and fibrotic remodeling (31, 32)

Cancer development and progression are closely associated with the processes of intracellular oxidative stress

On the one hand, transformation of TAMs reverses the suppressive tumor immune microenvironment and provides favorable conditions for antitumor immunity (Fig

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