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Since the characterization of disease-associated glial phenotypes in 2017, building on earlier pioneering discoveries, we focus here on disease-associated microglia (DAM) and disease-associated astrocyte (DAA) to reassess their contributions to glio-inflammation

(954) Khler, C

Total iron binding capacity rises

Correspondingly, we directly examined an established NRF2 gene expression signature (22) at each time point during LCS3 treatment in H23 and H1650 cell lines and observed an induction of NRF2-regulated genes (Fig