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Funding sources were as follows: DTR: R01GM115424 (NIH)

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Role of the tachykinin NK1 receptor in a murine model of cigarette smoke-induced pulmonary inflammation

As a consequence, a loss of lipoic acid-dependent enzymatic activity caused by defects in endogenous lipoic acid synthesis (see Deficiency) cannot be rescued by the provision of exogenous lipoic acid (5)