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Emerging cross-kingdom evidence indicates that redox-regulated mitochondrial proteostasis and calcium-dependent stress signalling modules identified in plant systems can inform mechanistic hypotheses in mammalian neurodegeneration

14 In addition to protecting the neonatal mice, it has been shown that overexpression of TRX-1 also reduced hyperoxia-induced acute lung injury in adult mice accompanied by reduced protein leakage into the lungs, alveolar damage, focal alveolar hemorrhage, and hyaline membrane deposits in the lungs