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YAP enhances TCA activity by increasing Lars2 expression, whereas ARID1A deletion forces metabolic flux toward the TCA cycle by suppressing PKM

A strategic approach to treating GBM involves exploiting the nuances of DNA repair mechanisms to create a situation where the repair capacity is overwhelmed by the rate of damage, thereby enhancing the efficacy of therapies [158]

Studies have shown that DSIP and its analogs are classified as peptide neuromodulators

Sustained microglial activation leads to the release of pro-inflammatory cytokines such as TNF-, IL-1, and IL-6, which exacerbate neuronal injury and amplify amyloid and tau pathology (Kaur et al., 2019)