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The critical role of PGRN and TMEM106B in brain aging is also supported by mouse genetic studies, where deletion of either Grn or Tmem106b gene is reported to cause accelerated accumulation of lipofuscin in the brain [88, 89]

Nielsen ML, Pareek M, Andersen I

The unveiling of SLC1A5, SLC7A5, SLC6A14, and the SLC38 family transporters, along with their distinctive functional characteristics, enriches our comprehension of the intricate nature of glutamine transport in cancer

UA improves cellular resilience by stimulating mitophagy, the selective autophagic removal of damaged mitochondria (Figure 1) (20)

While their overall trends showed no significant discrepancies in interpretation, from a methodological perspective, it is advisable to select identical brain regions for investigation