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Accumulating evidence indicates that AKI is not merely a transient hemodynamic disturbance but a biologically orchestrated process in which dysregulated intracellular signaling pathways translate ischemic stress into sustained renal injury and long-term functional decline

Depletion of intracellular GSH in HepG2 cells with BSO treatment was without effect on cell viability as assessed by MTT assay (Fig

This review consolidates recent findings indicating that FOXO4-dependent senescence significantly contributes to neuroinflammation, synaptic dysfunction, and impaired neurogenesis in the aging brain

A progressive structural understanding of the detailed receptor-ligand interactions is paving the way for fragment-to-lead optimization in structure-based drug design (SBDD) (Fig

Larger jumps risk triggering intolerance that wouldn't occur with gradual progression