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Description

Differential inhibition of cellular glutathione reductase activity by isocyanates generated from the antitumor prodrugs Cloretazine and BCNU Biochem

Methods Growth conditions, genetic manipulation, and strains Cells were grown in rich medium (YE) or minimal medium (MM) at 30 C as described previously [36]

Keap1 controls protein S-nitrosation and apoptosis-senescence switch in endothelial cells

Genetic polymorphisms of human N-acetyltransferase, cytochrome P450, glutathione-S-transferase, and epoxide hydrolase enzymes: relevance to xenobiotic metabolism and toxicity