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Although IF is linked to processes like autophagy, hormonal changes, and enhanced insulin sensitivity, the exact biological mechanisms that make IF safe and work are not fully understood

2 Ferroptosis and its link to DCM Ferroptosis is a form of programmed cell death driven by the excessive accumulation of iron ions within cells, triggering the Fenton reaction, activating oxidative stress, and disrupting lipid metabolism, ultimately leading to cellular demise ( Acyl-CoA synthetase long-chain family member 4 (ACSL4) is a key enzyme involved in the synthesis of polyunsaturated fatty acid phosphatidylethanolamine (PUFA-PE) ( In patients with DCM, the GSH-dependent antioxidant mechanism is suppressed, whereas ACSL4 activation promotes ferroptosis in myocardial cells ( In the development of DCM, sustained hyperglycemia acts as a key driver of ferroptosis

In patients with moderate or severe liver injury, prednisolone should be administered with discontinuation of the causative drug

106 This decreases hepatic glucose production and reduces hyperglycemia

The U/ml was calculated on the basis of the amount of H 2 O 2 reduced by Hb during the reaction, using g/ml and U/mg