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234 The nonsteroidal anti-inflammatory drug diclofenac can significantly reduce the protein expression level of NF-B p65 and the phosphorylation level of IB-, effectively protecting the structure and function of the intestinal barrier by suppressing the iNOS/NF-B pathway and activating the expression of HO-1

Our data indicated that 797 transcripts were upregulated, and 1383 transcripts were downregulated in ESCC compared with the control (Figure 2a and Supplementary Table 1)

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Similar content being viewed by others Facts Catalyzed by iron and relying on lipid peroxidation, ferroptosis is a type of cell death that has recently been studied in relationship to acute inflammatory and chronic fibrotic damage caused by radiation therapy