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Similarly, RAGE inhibition may inadvertently impair beneficial Treg subsets reliant on glycolytic intermediates for anti-inflammatory function [215]

We also observe that the levels of PINK1 are not substantially affected by PRT062607, implying that the observed reduction in pUb is not caused by a reduction in PINK1 levels

This is intriguing considering that Grx2 deglutathionylase activity operates via a monothiol mechanism which requires only the N-terminal active site cysteine

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