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A splicing switch from ketohexokinase-C to ketohexokinase-A drives hepatocellular carcinoma formation

Introduction Protection against oxidative stress (O.S.)induced damage is a common feature of every living organisms (recently reviewed by Siauciunaite et al [2019]) and is mediated by a wide range of biological processes including (but not limited to) glutathione-induced gene expression (Hayes & McLellan, 1999), and regulation of NADPH levels via the pentose phosphate pathways (Ralser et al, 2007) and the superoxide dismutase (Muid et al, 2014) and catalase (Chelikani et al, 2004) activity

(Kishneez) exhibited antianxiety, anti-epileptic, antioxidant, and anti-inflammatory properties

A high-purity preparation minimizes the risk of confounding signals from contaminants or degradation byproducts, giving your results a defensible baseline

If nothing changes, that is still useful information: it may mean the bottleneck is elsewhere, or the chosen form is not a good match